NOMOZ Mosquito Larvicide Pellets

1 Pack is 15g

NOMOZ Pellets prevent mosquito larvae developing into normal pupae by interfering with the growth messages the mosquito larvae receive. Death can occur either as larvae or pupae. Both larvae and pupae may still be seen after treating with NOMOZ pellets, however, these will eventually die and won't emerge to become biting adult mosquitoes that could spread diseases such as Ross River virus, Barmah Forest virus, dog heartworm and Dengue fever. 

The active ingredient in NOMOZ Pellets, (S)-methoprene, has been used in professional mosquito control programs for over 30 years. Pupae which are present before treatment will not be effected, therefore, NOMOZ Pellets should be applies before pupae are present. 

NOMOZ can be used in small, natural and artificial containers such as, but not limited to; Birdbaths, Pot Plant Holders, Roof Guttering and Ornamental Plants, such as Bromeliads. Used as directed, it will not adversely effect; people, fish, pets or vegetation. 

How To Use: Place pellet(s) into water container to be treated so that they can not be covered by sediment. Use 1 pellet per for m²  for control up to 3 months or use 3 pellets per for m²  for control up to 6 months.

After Application of NOMOZ?

• You may see living mosquito larvae. These may take over a week to die.
• You may see abnormally light coloured mosquito larvae and/or pupae.
• You may see slow or unresponsive mosquito larvae and/or pupae.
• You may see malformed mosquito larvae and/or pupae.

NOMOZ ® OVERVIEW

The active ingredient in NOMOZ® is PROLINK® which contains (S)-methoprene, an insect growth regulator (IGR). IGR’s are classified as biorational control agents. IGR’s control insects by interrupting normal development. (S)-methoprene interferes with normal metamorphosis, preventing the immature insect from emerging as an adult.

(S)-methoprene is a true analogue of the mosquito’s own juvenile hormone. During the fourth larval instar, juvenile hormone (JH) levels drop to very low levels. NOMOZ®, when applied into the larval habitat, releases (S)-methoprene which artificially maintains the JH levels at a higher than normal levels. The higher than normal level during the latter instar stages, prevents the insect from developing into normal pupa. Mortality occurs during the emergence of biting adult mosquitoes.

Unlike conventional pesticides, (S)-methoprene is not a direct toxin. It is target-specific and does not harm mammals, waterfowl, or even beneficial predatory insects. In fact, methoprene has been recommended by the World Health Organisation (WHO) for the use in drinking water supplies to control container-breeding mosquitoes in developing countries.

By working specifically on fourth instar mosquito larvae, NOMOZ® offers effective control without upsetting the food chain relationships between larvae and their natural predators. And, most importantly, without impacting non-target species.

Environmental Properties

General: (S)-methoprene insect growth regulator (IGR) is a biodegradable compound of very low environmental persistence. The (S)-methoprene molecule contains only carbon, hydrogen and oxygen.

Persistence in Soil: The half-life of methoprene when applied to soil at 1 Kg per Hectare is less than 10 days. This is a highly exaggerated application rate with regard to the intended use pattern.

Movement in Soil: In leaching studies, it has been shown that methoprene remains in the top 10 cm of the soil even after repeated washings with water.

Persistence in Water: Technical methoprene degrades rapidly when applied to water under normal conditions of sunlight and temperature. Methoprene is degraded by aquatic microorganisms and sunlight. Slow release and microencapsulated formulations allow its operational use for mosquito control.

Persistence in Plants: When applied at the rate of 1 Kg per Hectare to alfalfa, methoprene has a half-life of less than two days. In rice, the half-life is less than one day. Rapid biodegradation is evident even after treatment at exaggerated rates.

Uptake by Plants: Studies show that wheat grown in methoprene-treated soil does not contain residues of methoprene.

Fate in Animals: Laboratory and field experiments have shown that methoprene is rapidly degraded by mammalian metabolic systems. When mice, rats, guinea pigs, steers, or cows are treated orally with methoprene, the compound is metabolised quickly and eliminated via urine, faeces, and expired breath.

Fate in Fish: In model ecosystem experiments, methoprene is shown not to accumulate in fish and does not inhibit growth or development of fry.

Effects on Non-Target Organisms: Toxicity studies have been conducted both in the laboratory and as part of field research programs. The action of methoprene is selective. When applied at the recommended use rate, methoprene does not harm wild fowl, amphibians, crustaceans or beneficial insects.

NOMOZ Brochure